Andrew A. Dahl, MD, is a board-certified ophthalmologist. Dr. Dahl's educational background includes a BA with Honors and Distinction from Wesleyan University, Middletown, CT, and an MD from Cornell University, where he was selected for Alpha Omega Alpha, the national medical honor society. He had an internal medical internship at the New York Hospital/Cornell Medical Center.
As with other types of glaucoma, the frequency of follow-up care depends on the level of IOP control and the risk of vision loss.
If IOP is elevated soon after a blunt trauma, the patient may be reexamined every 4 to 6 weeks during the first year to monitor his or her condition. Although most cases do not progress to angle recession glaucoma, the patient should continue to be checked even after the condition has appeared to resolve. Sometimes, the early elevation of IOP represents a severe form of the disease that may not respond to standard medical treatment. Severe forms require more frequent follow-up care. Gonioscopy performed after the risk of bleeding is over will determine whether the angle is recessed.
In eyes with angle recession involving more than 180° of the iris but without any signs of glaucoma, the potential for late-onset glaucoma is still a reasonable concern, even many years after the injury. If a patient falls into this category, eye examinations will be performed at 6-month or yearly intervals for an indefinite period of time.
The incidence of angle recession glaucoma could be reduced by prevention of underlying trauma. Data indicate that most eye injuries (for
example, sports-related accidents) in both adults and children are preventable.
Public education on the use of eye, face, and/or head protection during high-risk activities may lower the incidence of eye injuries.
Public safety standards to reduce the rates of eye injuries can be achieved by enacting legislative policies, such as seatbelt laws and helmet laws.
Traumatic glaucoma refers to a heterogeneous group of posttraumatic ocular disorders with different underlying mechanisms that lead to the common pathway of abnormal elevation of intraocular pressure (IOP) and increased risk of optic neuropathy.