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Medical Author:

Siamak T. Nabili, MD, MPH

Siamak T. Nabili, MD, MPH

Dr. Nabili received his undergraduate degree from the University of California, San Diego (UCSD), majoring in chemistry and biochemistry. He then completed his graduate degree at the University of California, Los Angeles (UCLA). His graduate training included a specialized fellowship in public health where his research focused on environmental health and health-care delivery and management.

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Medical Editor:

Melissa Conrad Stöppler, MD, Chief Medical Editor

Melissa Conrad Stöppler, MD, Chief Medical Editor

Melissa Conrad Stöppler, MD, is a U.S. board-certified Anatomic Pathologist with subspecialty training in the fields of Experimental and Molecular Pathology. Dr. Stöppler's educational background includes a BA with Highest Distinction from the University of Virginia and an MD from the University of North Carolina. She completed residency training in Anatomic Pathology at Georgetown University followed by subspecialty fellowship training in molecular diagnostics and experimental pathology.

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Any therapy to lower CRP levels focuses on lowering the cardiovascular risk factors. Regular exercise, appropriate diet, and smoking cessation are in the forefront of cardiovascular risk prevention and reduction.

Cholesterol-lowering medications (statins) have been linked to lowering of CRP levels in individuals with high cholesterol. The fall of CRP levels may occur even without significant improvement in cholesterol levels.

The use of aspirin in healthy individuals was not shown to reduce CRP levels significantly. However, in patients with cardiovascular disease and elevated CRP, the reduction of cardiovascular risk and CRP levels was noted after aspirin use.

Some oral diabetes medicines, thiazolidinediones (rosiglitazone and pioglitazone), were noted to reduce CRP levels in patients with or without type 2 diabetes. This effect was independent of their glucose-lowering effects.

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