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May 24, 2013
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Occupational Asthma (cont.)

Medical Author:
George Schiffman, MD, FCCP

Dr. Schiffman received his B.S. degree with High Honors in biology from Hobart College in 1976. He then moved to Chicago where he studied biochemistry at the University of Illinois, Chicago Circle. He attended Rush Medical College where he received his M.D. degree in 1982 and was elected to the Alpha Omega Alpha Medical Honor Society. He completed his Internal Medicine internship and residency at the University of California, Irvine.

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Medical Editor:
William C. Shiel Jr., MD, FACP, FACR
William C. Shiel Jr., MD, FACP, FACR
William C. Shiel Jr., MD, FACP, FACR

Dr. Shiel received a Bachelor of Science degree with honors from the University of Notre Dame. There he was involved in research in radiation biology and received the Huisking Scholarship. After graduating from St. Louis University School of Medicine, he completed his Internal Medicine residency and Rheumatology fellowship at the University of California, Irvine. He is board-certified in Internal Medicine and Rheumatology.

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Occupational Asthma Prevention

Treatment in occupational asthma is focused on preventing or minimizing asthma attacks. The main strategy for doing this is reducing or stopping exposure to the trigger.

  • Work with your employer to "clean up" the workplace.


  • You or your health-care provider should be able to arrange for measurement of air quality in the workplace.


  • Your employer should provide protective gear, such as masks or respirators, to avoid exposure to the trigger.


  • Careless use or spills of respiratory irritants, inappropriate ventilation, and improper protective gear contribute to the occurrence of asthma in the workplace. These problems can be remedied.


  • If these measures don't reduce your symptoms, talk to your employer about retraining for a different position that would not involve exposure to your trigger.

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Allergic and Environmental Asthma »

Asthma is a clinical syndrome characterized by episodic reversible airway obstruction, increased bronchial reactivity, and airway inflammation.

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